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Health Effects and Symptoms Associated with Mold Exposure

 

 

 

Health Effects and Symptoms Associated with Mold Exposure

When moisture problems occur and mold growth results, building occupants may begin to report odors and a variety of health problems, such as headaches,  breathing difficulties, skin irritation, allergic reactions, and aggravation of asthma symptoms; all of these symptoms could potentially be associated with mold exposure.

All molds have the potential to cause health effects. Molds produce allergens, irritants, and in some cases, toxins that may cause reactions in humans. The types and severity of symptoms depend, in part, on the types of mold present, the extent of an individual's exposure, the ages of the individuals, and their existing sensitivities or allergies.

Research on mold and health effects is ongoing. This list is not intended to be all-inclusive.

The health effects listed above are well documented in humans. Evidence for other health effects in humans is less substantial and is primarily based on case reports or occupational studies.

 

Specific reactions to mold growth can include the following:

  • Allergic Reactions
    Inhaling or touching mold or mold spores may cause allergic reactions in sensitive individuals. Allergic reactions to mold are common - these reactions can be immediate or delayed. Allergic responses include hay fever-type symptoms, such as sneezing, runny nose, red eyes, and skin rash (dermatitis). Mold spores and fragments can produce allergic reactions in sensitive individuals regardless of whether the mold is dead or alive. Repeated or single exposure to mold or mold spores may cause previously non-sensitive individuals to become sensitive. Repeated exposure has the potential to increase sensitivity.
  • Asthma
    Molds can trigger asthma attacks in persons who are allergic (sensitized) to molds. The irritants produced by molds may also worsen asthma in non-allergic (non-sensitized) people.
  • Hypersensitivity Pneumonitis
    Hypersensitivity pneumonitis may develop following either short-term (acute) or long-term (chronic) exposure to molds. The disease resembles bacterial pneumonia and is uncommon.
  • Irritant Effects
    Mold exposure can cause irritation of the eyes, skin, nose, throat, and lungs, and sometimes can create a burning sensation in these areas.
  • Opportunistic Infections
    People with weakened immune systems (i.e., immune-compromised or immune-suppressed individuals) may be more vulnerable to infections by molds (as well as more vulnerable than healthy persons to mold toxins). Aspergillus fumigatus, for example, has been known to infect the lungs of immune-compromised individuals. These individuals inhale the mold spores which then start growing in their lungs. Trichoderma has also been known to infect immune-compromised children.

    Healthy individuals are usually not vulnerable to opportunistic infections from airborne mold exposure. However, molds can cause common skin diseases, such as athlete's foot, as well as other infections such as yeast infections.

 

Why is there controversy about the health effects of exposure to mold growth? Not all health effects of molds are controversial. Fungal infections are well known. Fungal allergies are also well known and accepted among medical experts, although the allergens themselves are poorly characterized. Infections and allergies have objective and well-established clinical effects. These effects can be measured and reproducibly demonstrated, and the mechanisms are fully understood. The health effects caused by consuming moldy food or feed that contains mycotoxins are also well known. Regardless of these controversies, mold growth in the built environment is unacceptable from the perspectives of potential adverse health effects and building performance.

Other health effects have been proposed for mold metabolites that are irritants or mycotoxins, and plausible mechanisms exist for health effects due to these mold metabolites. However, the clinical relevance of these mycotoxins and irritants under realistic airborne exposure levels is not fully established. Further, supporting evidence for other health effects is based on case studies rather than controlled studies, nonreproduced studies, or subjective symptoms.

Case studies do indicate the possibility or plausibility of an effect. Unfortunately, such studies cannot address whether an effect is common or widespread among building occupants. Results from nonreproduced studies may be false or are not confirmed by well-designed follow-up studies. In large epidemiological studies, general symptoms have been associated with moisture-damaged and presumably moldy buildings. Many of the reported symptoms are subjective and difficult to quantify. Results are confounded by the fact that the association is general, and mold is not the only possible cause of the symptoms. Neither condition proves that mold is not a cause.

Since much remains unproven, controversy has developed around the presumed health effects attributed to mycotoxins. This controversy is intensified since the health effects are often serious and sometimes are claimed to be permanent. Dampness in buildings is associated with respiratory effects, but the extent to which mold contributes to these effects is unknown. Some health effects from mold exposure remain controversial because of the potentially significant consequences; yet crucial and legitimate scientific questions remain unanswered. Our incomplete knowledge of noninfectious health effects related to mold exposure is due to limited research support and lack of documented health effects in the context of well-defined exposures. Bear in mind that environmental investigations must also be interpreted in context with medical and epidemiological information for infectious diseases from environmental sources. For example, finding Legionella colonization of a water supply serving an immunocompromised population would have potential health significance whether or not there were cases of infection, but finding mold spores in ambient air has little significance unless people are getting sick from direct exposure.

Why are there no standards for mold exposure? Health hazards from exposure to environmental molds and their metabolites relate to four broad categories of chemical/biological attributes. These materials may be: 1) irritants, 2) allergens, 3) toxins, and rarely 4) pathogens. Different mold species may be more or less hazardous with respect to any or all of these categories. However, risks from exposure to a particular mold species may vary depending on a number of factors. Uncertainty is complicated further by a lack of information on specific human responses to well-defined mold contaminant exposures. In combination, these knowledge gaps make it impossible to set simple exposure standards for molds and related contaminants.

 

What are the knowledge gaps concerning mold exposure and its health effects? Chief among our knowledge gaps are: (1) defining how mycotoxins affect human health and (2) the health risks associated with mycotoxin, microbial volatile organic compound, allergen, and glucan exposures, particularly the proposed response to Stachybotrys mycotoxins associated with hemosiderosis.

However, the etiology of infectious fungi is relatively well understood. Conversely, mechanisms responsible for allergic sensitization, contact dermatitis, hypersensitivity pneumonitis, and inhalation fevers vary from incompletely characterized to entirely unknown. Predisposing host factors, presumably under genetic control, influence individual susceptibility to environmental exposures. The psychogenic/psychosocial contribution to mold-related illness remains elusive.

The lack of meaningful exposure limits for most indoor air quality contaminants is a major obstacle to establishing regulatory standards for individual exposure to airborne contaminants. The same is certainly true for molds. Until microbiological methods for demonstrating mold concentrations in the environment are standardized and reproducible, epidemiological studies necessary to determine dose-response can only suggest association, not cause and effect, with respect to mold exposures and health effects.